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IGFBPs can also act to guide IGF-1 to specific tissues, or even to inhibit or potentiate IGF-1 actions by acting as an independent substrate for the IGF-1R and/or other specific membrane, intracellular or nuclear receptors [9395]

This process is crucial for preventing the accumulation of damaged mitochondria, which can compromise cellular energetics and contribute to age-related decline

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Journal of Pharmacy and Nutrition Sciences, 2024

GHK-CU works through gene expression and copper metabolism